By Adrianne Tillman in Open Medicine Foundation.
Leading scientists presented their research on ME/CFS at the day-long Community Symposium on the Molecular Basis of ME/CFS* at Stanford on Aug. 12. The researchers spoke at length about the complicated biological processes involved, their study results and the limitations of their conclusions presented thus far.
The amount of science on ME/CFS presented was overwhelming, and the article below highlights only a few of the studies discussed.
*ME/CFS was the terminology used at the conference.
The Mighty Mitochondria
The miraculous mitochondria was the headliner at the Community Symposium on the Molecular Basis of ME/CFS, which was recorded via livestream. Each researcher attempted to break it down for the layperson but the take-away was clear: something is going on with the mitochondria of people with Myalgic Encephalomyelitis (also known as Chronic Fatigue Syndrome) or ME/CFS.
Bob Naviaux, MD, PhD from the University of California, San Diego (UCSD) took the first stab at explaining the mitochondria as it relates to the “healing cycle” and the Cell Danger Response.
The mitochondria has two roles: energy production and cell defense, Naviaux explained.
“The mitochondria coordinates our cellular defense system. Think of them as concert masters. When they encounter danger, they send out danger signals that cause the cell to change its function from daily housekeeping functions to defending itself.”
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